Stress increases monoamine levels and increases smoking. Individuals that have a genetic variant that metabolizes monoamines slower have an greater increase in smoking following stress.
-
Slower Monoamine Metabolism Following Stress Increases Smoking
Skip to
CURRENT LESSON OBJECTIVES
- What is the heritability for nicotine dependence relative to other drugs of abuse?
- What is the Evidence for Genetic Factors in Smoking Initiation and Heavy Smoking?
- How Could Prevalence of Smoking Influence Estimates of Heritability?
- How do the Genetics of Nicotine Metabolism Relate to Smoking and Treatment ?
- How do variations in genes for acetylcholine receptors relate to smoking behaviors ?
- How does the Alpha 5 Receptor subunit Interact with Risk Factors for Smoking?
- How Does Stress Interact with Genetics of Monoamine Metabolism to Increase Smoking?
- How Would a Dopamine Receptor Allele Associated with Alcohol Addiction Effect Smoking ?
- How Could Prenatal Nicotine Exposure Alter Dopaninergic Function in Adulthood?
- Could Genotype Help Predict Response to Treatment?
RELATED RESOURCES
Related Clinical Cases
- Adolescent Drinking (Sneaky Teen is Not Squeeky Clean)
- Alcohol Withdrawal focuses on
- Binge Drinking in College (Rebel Without a Cause)
- Fetal Alcohol Syndrome (FAS) focuses on
Related Quizzes
- Alcohol and accidents focuses on
- Alcohol and adolescence focuses on
- Alcohol Genetics focuses on
- Alcohol withdrawal focuses on
- Diagnosis and treatment focuses on
- Epidemiology
- Fetal alcohol syndrome focuses on
- Medical complications
- Neurobiology focuses on
- Pharmacology and acute effects alcohol
- Regular drinking focuses on